Long PR Interval

The PR interval is the time from the beginning of the atrial wave to the beginning of myocardial depolarization of the ventricles. Normally this takes 0.12 – 0.20 seconds. When the time taken is over 0.20 seconds, this is called a long PR interval.

Following the wave of atrial depolarization which produces a P wave on an ECG, is the time for the impulse to proceed across two small structures – the AV node and the bundle of His – that connect the atria to the ventricles. Like the SA node, these two structures – called the AV junction – are too small to generate enough voltage to produce an electrical wave. The time taken to cross these structures are captured in the PR line.

The expected conduction speed measured from the beginning of atrial depolarization (beginning of the P wave) to the beginning of the ventricular depolarization (beginning of the QRS complex) is captured in the PR interval. This is typically .12-.20 seconds (3-5 mm on a rhythm strip).

A long PR interval is usually caused by a longer PR line than a wider P wave that is longer. The PR line is most reflected from the time taken for the impulse to cross the AV junction. For a PR interval longer than .20 seconds, consider how and why this is so. Might a medication be having considerable effect on nodal conduction speed (ABCD – amiodarone, beta blocker, calcium channel blocker, digoxin)? Might there be nodal structural disease or ischemia effecting the area? Occasionally, this is caused simply because a person has a larger heart – larger person or enlarged heart – that takes a little longer for an impulse to cross. This can also be an unexplained phenomena as well.

Atrioventricular blocks (AV blocks) result from a conduction disturbance at or just below the AV junction. The 3rd step of the 3 step process prompts us to check the P waves and Pattern with a focus on the PR interval. Abnormal PR intervals and lonely ‘P’ waves define the type of AV block.

From a clinical perspective, the severity of a block is similar to the severity of burns. The higher the degree of burn the more aggressive the treatment. Similar escalation in treatment is required for higher levels of AV blocks. The affects of 2nd degree type II and 3rd degree AV blocks on cardiac output can be much more significant than the affects of 2nd degree type I and 1st degree AV blocks.

First degree AV block is simply conducts slower through the AV junction. First degree AV block can be a benign finding (particularly athletes). Other causes include ischemia, increased vagal tone, and the effects of medications that slow conduction across the AV junction: digoxin, calcium channel blockers, and beta blockers for example. As a result of the slowed junctional conductivity, the PR interval of first degree AV block is longer than normal (> 5 mm in width).

The significance of 1st degree block is revealed in its position in the naming of a rhythm. The underlying rhythm is identified first followed by descriptors of any abnormal components such as 1st degree AV block.

sinus tachycardia, HR 102/min, long PR, wide QRS, PVCs, JEC, short PR intervals

1. Six Second ECG Guidebook (2012), T Barill, p. 125-126, 189

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